Researchers at the Terasaki Institute for Biomedical Innovation have conducted a groundbreaking study exploring the relationship between amyloid-like structures and neuronal cells in Alzheimer’s disease. By creating a self-assembling peptide-based hydrogel that mimics the amyloid-beta environment associated with AD, they were able to observe elevated neuroinflammation and apoptosis markers in healthy neuronal cells. The study sheds light on the complex mechanisms underlying AD and provides a promising model for future research and drug testing. This innovative approach bridges the gap between traditional 2D models and the intricate reality of AD, offering new insights into potential therapeutic strategies.
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New 3D model sheds light on amyloid-beta’s impact
