Researchers find genetic variant coding for tubulin protein that may be partially responsible for left-handedness

A study published in Nature Communications by researchers from the Max Planck Institute for Psycholinguistics identified a genetic variant related to left-handedness by analyzing data from over 300,000 individuals in the UK Biobank. The study found that left-handed individuals were more likely to have a rare genetic variant affecting tubulin protein, which plays a role in cell shape and neuron development. Previous research has linked tubulin to hemisphere dominance, suggesting a potential connection between microtubule development and left-handedness. This finding adds to the understanding that left-handedness is likely influenced by a combination of genetic factors.

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