A study from Hebrew University’s Hadassah Medical School introduces a new model suggesting that disrupted RNA editing in pancreatic beta cells may trigger an inflammatory response similar to early-stage type 1 diabetes. This challenges the long-held belief of viral involvement and could have implications for T1D treatments and cures. This perspective offers a new insight into the disease without needing viral infection as the trigger, and introduces an alternative paradigm for T1D development. The research implies that defects in RNA editing may contribute to the onset of T1D and opens up new possibilities for prevention and treatments. The findings could significantly impact the way T1D is approached and understood.
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New model links RNA editing glitch to early-stage type 1 diabetes
